• Static Crackling On Headphones
• Stasis Cracks

Stasis dermatitis is a change in the skin that results for the pooling of blood in the veins of the lower leg. Ulcers are open sores that can result from untreated stasis dermatitis. This photograph shows a red to brownish discoloration just above the ankle, known as stasis dermatitis. It results from decreased blood flow. The Duskfield grenade with Stasis would slow down enemies and freeze them (sometimes repeatedly of you crack them and the grenade is still present). What if they used the grenade on Atraks? This is something that has been bugging me since last night.

Stasis dermatitis, also known as gravitational dermatitis, venous eczema, or venous stasis dermatitis, is a chronic inflammatory skin disease. It is caused because of the pooling of blood in the legs. Stasis dermatitis occurs due to poor blood flow, particularly in the lower limbs. Who gets stasis dermatitis? STASIS is a point-and-click, sci-fi - horror adventure game played from a unique isometric perspective. STASIS intertwines a gruesome narrative with retro adventure gameplay. Combine items, interact with computers and solve puzzles while experiencing the gritty atmosphere. Get the Stasis Rune. When you are in the shrine, check on the Guidance Stone to get the Stasis Rune added to your Sheikah Slate. How to Use the Stasis Rune. When you have selected the Stasis Rune on your slate, press L to start it. Then aim the reticle at anything that is glowing bright yellow. Press A to use stasis on that object.

What are ulcers?

Ulcers are wounds or open sores that will not heal or keep returning.

What causes leg ulcers?

Leg ulcers may be caused by medical conditions such as:

• Venous insufficiency (a failure of the valves in the veins of the leg that causes congestion and slowing of blood circulation in the veins)
• Diabetes
• Renal (kidney) failure
• Hypertension (treated or untreated)
• Lymphedema (a buildup of fluid that causes swelling in the legs or feet)
• Inflammatory diseases including vasculitis, lupus, scleroderma or other rheumatological conditions
• Other medical conditions such as high cholesterol, heart disease, high blood pressure, sickle cell anemia, bowel disorders
• History of smoking (either current or past)
• Pressure caused by lying in one position for too long
• Genetics (ulcers may be hereditary)
• A malignancy (tumor or cancerous mass)
• Infections
• Certain medications

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What are the types of leg and foot ulcers?

The three most common types of leg and foot ulcers include:

• Venous stasis ulcers
• Neurotrophic (diabetic)
• Arterial (ischemic ulcers)

Stasis Cracks

Ulcers are typically defined by the appearance of the ulcer, the ulcer location, and the way the borders and surrounding skin of the ulcer look.

Venous Stasis Ulcers

• Location onbody: Below the knee - primarily found on the inner part of the leg, just above the ankle. Ulcers may affect one or both legs.
• Appearance:
  • Base: Red in color and may be covered with yellow fibrous tissue. There may be a green or yellow discharge if the ulcer is infected. Fluid drainage can be significant.
  • Borders: Usually irregularly shaped. The surrounding skin is often discolored and swollen. It may even feel warm or hot. The skin may appear shiny and tight, depending on the amount of edema (swelling).
• Who is affected: Venous stasis ulcers are common in patients who have a history of leg swelling, varicose veins, or a history of blood clots in either the superficial or the deep veins of the legs. Venous ulcers affect 500,000 to 600,000 people in the United States every year and account for 80 to 90 percent of all leg ulcers.

Neurotrophic (diabetic) Ulcers

• Location onbody: Usually located at increased pressure points on the bottom of the feet. However, neurotrophic ulcers related to trauma can occur anywhere on the foot.
• Appearance:
  • Base: Variable, depending on the patient's circulation. It may appear pink/red or brown/ black.
  • Borders: Punched out, while the surrounding skin is often calloused.
• Whois affected: Neurotrophic ulcers occur primarily in people with diabetes, although they can affect anyone who has an impaired sensation of the feet.

Neuropathy and peripheral artery disease often occur together in people who have diabetes. Nerve damage (neuropathy) in the feet can result in a loss of foot sensation and changes in the sweat-producing glands, increasing the risk of being unaware of foot calluses or cracks, injury or risk of infection. Symptoms of neuropathy include tingling, numbness, burning or pain.

It is easy to understand why people with diabetes are more prone to foot ulcers than other patients. This is why people with diabetes need to inspect their feet daily and wear appropriate footwear. People with diabetes should never walk barefoot.

Arterial (ischemic) Ulcers

• Location onbody: On the feet - often on the heels, tips of toes, between the toes where the toes rub against one another or anywhere the bones may protrude and rub against bed sheets, socks or shoes. They also occur commonly in the nail bed if the toenail cuts into the skin or if the patient has had recent aggressive toe nail trimming or an ingrown toenail removed.
• Appearance:
  • Base: Has a yellow, brown, grey or black color and usually does not bleed.
  • Borders: The borders and surrounding skin usually appear punched out. If irritation or infection are present, there may or may not be swelling and redness around the ulcer base. There may also be redness on the entire foot when the leg is dangled; this redness often turns to a pale white/yellow color when the leg is elevated. Arterial ulcers are typically very painful, especially at night. The patient may instinctively dangle his/her foot over the side of the bed to get pain relief.
• Who is affected: The patient usually has prior knowledge of poor circulation in the legs and may have an accompanying disorder, such as those listed in the section, 'What causes leg ulcers?'

What are the symptoms of ulcers?

Ulcers may or may not be painful. The patient generally has a swollen leg and may feel burning or itching. There may also be a rash, redness, brown discoloration or dry, scaly skin.

Stasis dermatitis is a change in the skin that results for the pooling of blood in the veins of the lower leg. Ulcers are open sores that can result from untreated stasis dermatitis.

Venous insufficiency is a long-term (chronic) condition in which the veins have problems sending blood from the legs back to the heart. This may be due to damaged valves that are in the veins.

Some people with venous insufficiency develop stasis dermatitis. Blood pools in the veins of the lower leg. Fluid and blood cells leak out of the veins into the skin and other tissues. This may lead to itching and inflammation, which cause more skin changes. The skin may then break down to form open sores.

You may have symptoms of venous insufficiency including:

• Dull aching or heaviness in the leg
• Pain that gets worse when you stand or walk
• Swelling in the leg

At first, the skin of the ankles and lower legs may look thin or tissue-like. You may slowly get brown stains on the skin.

The skin may become irritated or crack if you scratch it. It may also become red or swollen, crusted, or weepy.

Over time, some skin changes become permanent:

• Thickening and hardening of the skin on the legs and ankles (lipodermatosclerosis)
• A bumpy or cobblestone appearance of the skin
• Skin turns dark brown

Skin sores (ulcers) may develop (called a venous ulcer or stasis ulcer). These most often form on the inside of the ankle.

The diagnosis is primarily based on the way the skin looks. Your health care provider may order tests to examine the blood flow in your legs.

Stasis dermatitis can also be related to heart problems or other conditions that cause leg swelling. Your provider may need to check your general health and order more tests.

Your provider may suggest the following to manage the venous insufficiency that causes stasis dermatitis:

• Use elastic or compression stockings to reduce swelling
• Avoid standing or sitting for long periods of time
• Keep your leg raised when you sit
• Try varicose vein stripping or other surgical procedures

Some skin care treatments can make the problem worse. Talk with your provider before using any lotions, creams, or antibiotic ointments.

Things to avoid:

• Topical antibiotics, such as neomycin
• Drying lotions, such as calamine
• Lanolin
• Benzocaine and other products meant to numb the skin

Treatments your provider may suggest include:

• Unna boot (compressive wet dressing, used only when instructed)
• Topical steroid creams or ointments
• Oral antibiotics
• Good nutrition

Stasis dermatitis is often a long-term (chronic) condition. Healing is related to the successful treatment of the cause, factors causing the ulcer, and prevention of complications.

Complications of stasis ulcers include:

• Bacterial skin infections
• Infection of bone
• Permanent scar
• Skin cancer (squamous cell carcinoma)

Call your provider if you develop leg swelling or symptoms of stasis dermatitis.

Watch for signs of infection such as:

• Drainage that looks like pus
• Open skin sores (ulcers)
• Pain
• Redness

To prevent this condition, control the causes of swelling of the leg, ankle, and foot (peripheral edema).

Venous stasis ulcers; Ulcers - venous; Venous ulcer; Venous insufficiency - stasis dermatitis; Vein - stasis dermatitis

Baxi O, Yeranosian M, Lin A, Munoz M, Lin S. Orthotic management of neuropathic and dysvascular feet. In: Webster JB, Murphy DP, eds. Atlas of Orthoses and Assistive Devices. 5th ed. Philadelphia, PA: Elsevier; 2019:chap 26.

Fitzpatrick JE, High WA, Kyle WL. Necrotic and ulcerative skin disorders. In: Fitzpatrick JE, High WA, Kyle WL, eds. Urgent Care Dermatology: Symptom-Based Diagnosis. Philadelphia, PA: Elsevier; 2018:chap 14.

Marks JG, Miller JJ. Ulcers. In: Marks JG, Miller JJ, eds. Lookingbill and Marks' Principles of Dermatology. 6th ed. Philadelphia, PA: Elsevier; 2019:chap 19.

Marston W. Venous ulcers. In: Almeida JI, ed. Atlas of Endovascular Venous Surgery. 2nd ed. Philadelphia, PA: Elsevier; 2019:chap 20.

Updated by: Michael Lehrer, MD, Clinical Associate Professor, Department of Dermatology, University of Pennsylvania Medical Center, Philadelphia, PA. Review provided by VeriMed Healthcare Network. Also reviewed by David Zieve, MD, MHA, Medical Director, Brenda Conaway, Editorial Director, and the A.D.A.M. Editorial team.